Moreover, through bioinformatic analysis, they found that stroke-related genes vascular endothelial growth factor-A (VEGFA), myeloid cell leukemia-1 (Mcl-1), and superoxide dismutase 2 (SOD2) might be the targets of miR-106b, therefore authors suggested that miR-106b may affect multiple pathways such as apoptosis, oxidation, angiogenesis, and neurogenesis in IS [68,69,70,71]. This evidence concerns the gene MCL1 and stroke disorder.