In silico analysis demonstrated that miR-106b-5p promotes the progression of cervical cancer by modulating the expression of glycogen synthase kinase 3 beta (GSK3B), vascular endothelial growth factor A (VEGFA), and protein tyrosine kinase 2 (PTK2) genes, which are genes that play a crucial role in phosphatidylinositol 3-kinase (PI3K)-Akt signaling and focal adhesion [57]. This evidence concerns the gene PTK2 and cervical carcinoma.