The rightward shift of the steady‐state inactivation curve may be one of the causes for a marked increase of whole‐cell Cav1.2 currents in HF offspring, while it did not contribute to the difference of the Cav1.2 currents between HHF and CHF cells, because the inactivation curve was similar between the two groups. This evidence concerns the gene CACNA1C and congestive heart failure.