Differential protein expression profiles between benign control and crocidolite-treated Met-5A mesothelial cells revealed activation of pathways related to DNA damage repair and cell cycle regulation and comparison between Met-5A and MM cells (NCI-H28) showed increased activation of the EGFR/ERK and PI3K/AKT pathways [28]. The gene discussed is EGFR; the disease is Miyoshi myopathy.