IFNG and Autoimmunity: In support of this eventuality are the findings demonstrating that an initial DC‐NK interaction controls an adaptive Th17 response and limits tissue‐specific autoimmunity through an innate IFN‐γ‐IL‐27 axis.9 Our data provide support for this possibility, as indicated by the presence of a regulatory role of T‐cell activation through DC‐NK interactions in EAU.