The balance between Interferon-alpha and beta at one side and gamma at the other (INF-α and β, and IFN-γ) activation in inflammatory processes can modulate the activation of KP and the resulting intensity of this inflammatory process, not just in SS but also in response to other stimuli, such as exogenous challenges [156,157,158]. This evidence concerns the gene IFNA17 and synovial sarcoma.