A number of studies have indicated that inhibition of LOX family members, in particular LOXL2, can positively influence myocardial remodelling,33, 34 with LOXL2 mRNA expression being highly upregulated upon cardiac disease.34 As such, the impact of LOXL2/LOXL3 enzymatic inhibition was studied in a model of post‐myocardial infarction remodelling. This evidence concerns the gene LOXL2 and myocardial infarction.