This is illustrated by studies demonstrating that, in contrast to our findings, KMT2C & KMT2D (Lysine methyltransferase 2C & D; also known as MLL3 and MLL2 respectively) actually promote nascent strand degradation in the presence of impaired fork protection (e.g. BRCA2-deficient tumour cells)4. This evidence concerns the gene KMT2C and neoplasm.