Abnormally activated EGFR drives the malignant phenotype including cell proliferation, survival, invasion and migration.13 Although several lncRNAs have been reported to modulate tumour proliferation, apoptosis or metastasis, the specific mechanisms of lncRNA involvement in EGFR signaling pathway‐mediated tumour progression of NSCLC are unclear. This evidence concerns the gene EGFR and non-small cell lung carcinoma.