The idea that dysfunction of autophagy is involved in vitiligo pathogenesis is supported by Jeong et al., who found that variation in UVRAG gene contributes to the risk of non-segmental vitiligo in the Korean population (50), and by Wang et al., who demonstrated that several genes involved in the autophagy process were dysregulated in leukocytes of generalized vitiligo patients (51). This evidence concerns the gene UVRAG and vitiligo.