For example, miR-155 promotes the expansion of pathogenic Th17 cells and further exacerbate the development of uveitis [35], while inhibition of miR-21 is correlated to the improvement of inflammatory BD-like symptoms through regulating cytokine expression and TLR4 [36], and hsa-miR-9 inhibits sympathetic ophthalmia through targeting TNF-α and nuclear factor kappa B1 [37]. Here, TNF is linked to uveitis.