IL17A and Miyoshi myopathy: As reported in human MM plasma cells31, Vk*MYC plasma cells (Supplementary Fig. 6) also expressed both subunits of the IL-17R (Fig. 3b), which was functional because exposure to saturating amounts of recombinant IL-17 induced STAT3 phosphorylation in Vk*MYC plasma cells, similarly to saturating amounts of IL-6 (Fig. 3c, e).