Type 1 ovarian cancers, which consist of low-grade serous carcinoma (LGSC), mucinous carcinoma, endometrioid carcinoma, malignant Brenner tumor, and clear cell carcinoma typically display a variety of somatic sequence mutations in KRAS, BRAF, PTEN, PIK3CA CTNNB1, ARID1A, and PPPWR1A, but very rarely in TP53 [4–6]. This evidence concerns the gene KRAS and ovarian carcinoma.