Increased VEGF expression is known to promote tumor growth, invasion, and metastasis.[11] VEGF levels were shown to be remarkably higher in TNBC than in non-TNBC.[12] Inhibiting the VEGF could reverse the enhanced migratory and invasive abilities of human TNBC cells.[13] These results suggest that anti-angiogenesis may pave a new way for the treatment of TNBC. Here, VEGFA is linked to neoplasm.