Biolayer interferometry analyses suggested that influenza strain A/mallard/Wisconsin/A00751454/2009(H1N1) had detectable binding affinities to 3ʹ-sialyl-N-acetyllactosamine (3ʹSLN, representing avian-like receptor SA2,3Gal) and to 6ʹ-sialyl-N-acetyllactosamine (6ʹSLN, representing human-like receptor SA2,6Gal). This evidence concerns the gene STAG2 and influenza.