Observations from the past decade (for a review see, Brahmachari et al., 2017) indicate that impaired activity of c-Abl is implicated in the pathogenesis of Parkinson’s disease (PD), a neurodegenerative disorder caused by the progressive loss of dopamine (DA)-producing cells in the substantia nigra (Lotharius and Brundin, 2002; Dauer and Przedborski, 2003; Lang and Espay, 2018). This evidence concerns the gene ABL1 and Parkinson disease.