Infection with Dengue and Zika viruses causes mitochondrial elongation, which also blocks MAVS function.128 The sustained interferon production caused by MAVS signalling from the peroxisomes is inhibited by the MIA protein from human cytomegalovirus (hCMV) and the VP16 protein from HSV‐1.118, 129, 130. The gene discussed is MAVS; the disease is infection.