Results suggested that not only did pretreatment with BI1002494 reduce infract size without bleeding, but also administration of the inhibitor after the stroke was generated had similar benefits, providing a compelling case for continued investigation and translation of SYK inhibition strategy to human patients.28 Inspired by our previous findings and the protective role of SYK inhibition in atherosclerosis‐related diseases, we have been suggested that SYK inhibition might be partially regulated by miRNAs. The gene discussed is SYK; the disease is atherosclerosis.