Taken together, these data indicate that both p53 null and mutant p53-expressing tumour cells release exosomes in similar quantities, but those from mutant p53-expressing cells upregulate RCP and DGKα-dependent receptor recycling in p53 null recipient cells to evoke migratory characteristics associated with mutant p53’s invasive gain-of-function. This evidence concerns the gene RAB11FIP1 and neoplasm.