Following infection, we examined virus replication-driven GFP expression (Fig. 9B), collected cell supernatants at different time points, and measured the production of the three most important antiviral IFNs, IFN-α (type I IFN), IFN-β (type I IFN), as well as IFN-λ2 (also known as interleukin-28A [IL-28A]), a type III IFN. Here, IFNB1 is linked to infection.