CCL2 and renal fibrosis: Based on both in vivo and in vitro results we propose that PAR‐1 potentiates renal fibrosis by regulating the expression of pro‐fibrotic mediators MCP‐1 and TGF‐β subsequently leading to MCP‐1‐induced macrophage influx and TGF‐β‐induced extracellular matrix production.