Next to its role in EMT, PAR‐1 has also been implicated in the production of pro‐fibrotic and/or pro‐inflammatory mediators, like MCP‐1 and TGF‐β, during pulmonary fibrosis.13, 26 To determine whether PAR‐1 would play a role in the production of these pro‐fibrotic and/or pro‐inflammatory mediators in the setting of renal fibrosis as well, we assessed PAR‐1‐induced cytokine production by TECs in vitro. Here, CCL2 is linked to pulmonary fibrosis.