The RNAi of BuGZ in several human cell lines resulted in chromosomal misalignment and mitotic block in the M phase.39 KIFC3 and KIF3b, members of the human kinesin family, have been reported to be highly expressed in seminoma tissues; the proteins also bind along the spindle and affect spindle formation in Hela cells.40, 41 Given these data, we conclude that CSE1L is essential to proper cell division and the disrupted CSE1L expression in TCam‐2 cells led to M phase delay. The gene discussed is KIFC3; the disease is seminoma.