NTRK1 and neoplasm: In contrast, K response was not associated with immune pathways, but the tumours in K responders (K+) versus non-responders (K−), were positively enriched in pathways that were related to extracellular matrix (ECM) organisation and receptor tyrosine kinase (RTK) signalling and negatively enriched for TCA- and proliferation-related genes (Fig. 1b and Table S2).