ADSL and glioblastoma: Many of the AMPs, with the inherent characteristics of being linear, cationic and amphipathic, have been proved to exert antiproliferative capacities through non‐specific interaction with eukaryotic cell membranes,23 while internalisation initiated apoptosis induction has also been reported to act as the major mechanism of anticancer functions.24 Thus, we tested the antiproliferative activity of Dermaseptin‐PS1 using different cell lines, and the results (Figure 3 and Figure S2) indicated that the glioblastoma U‐251 MG cells have lowest survival rates after Dermaseptin‐PS1 treatment.