2015; Park, Min et al. 2015, 2016). On the contrary, NPY treatment promoted neuroprotection and restored sensory neuropathy, HSC failure, and cell damage from chemotherapy in bone marrow microenvironment, suggesting the therapeutic potential of NPY against chemotherapy-induced bone marrow dysfunction (Park, Jin et al. 2015; Park, Min et al. 2015). The gene discussed is NPY; the disease is Sensory neuropathy.