HuR stabilizes ARE‐mRNAs involved in multiple biological events such as carcinogenesis, differentiation, and inflammation.15, 16 HuR is overexpressed in cancers, such as in oral, colon, ovarian, brain, breast, and pancreatic cancers.17, 18, 19, 20, 21, 22, 23 In particular, the abundant cytoplasmic expression of HuR is associated with the malignant phenotype of several types of carcinomas.19 We previously reported that HuR knockdown attenuated the malignant phenotype by inhibiting the stabilization of ARE‐mRNA. Here, ELAVL1 is linked to carcinoma.