showed that cell division cycle 5‐like protein (CDC5L) is a regulatory target of NEAT1 and that knockdown of NEAT1 results in reduced expression of AGRN, which is a direct target of CDC5L and a interaction partner of transforming growth factor beta 1 (TGFβ1) leading to DNA damage, cell cycle arrest ultimately resulting in decreased cancer cell growth and tumorigenesis in prostate cancer cells (Li et al., 2018b). This evidence concerns the gene CDC5L and cancer.