LMNA and congenital muscular dystrophy: Nuclear translocation of MKL1 was inefficient in LmnaΔ8−11/Δ8−11, Emd−/y, and LmnaN195K/N195K MEF as well as in patient skin fibroblasts with LMNA mutation leading to isolated cardiomyopathy (LMNA p.K219T) following serum stimulation, while it was not altered in another patient's skin fibroblasts with LMNA mutations leading to congenital muscular dystrophy (LMNA p.K32del) (Ho et al., 2013a,b).