Tumor-specific patterns of PPARγ expression support that PPARγ is strongly involved in maintaining homeostatic processes by adapting lipid and carbohydrate metabolism to respective tumor specific conditions, and by controlling tumor suppressor gene expression for keeping homeostatic pathways under tumor growth-promoting conditions, such as Wnt, Hippo-YAP pathway, etc. [27,39,40,41]. Here, PPARG is linked to neoplasm.