In contrast to IgE-mediated food allergy, in which sensitisation to a limited number of ‘informative’ allergenic proteins differentiates between true food allergy and asymptomatic sensitisation (such as Ara h 2 in peanut allergy) [9], we did not identify such ‘informative’ component(s) as a hallmark of an increased risk of asthma. The gene discussed is IGHE; the disease is peanut allergic reaction.