Previous studies demonstrated that DMY suppressed cell proliferation, induced cell cycle arrest at G1/S phase and promoted apoptosis of melanoma SK-MEL-28 cells, which might be related to the enhanced expression of Bax proteins and decreased levels of IKK-α, NF-κB, and P-p38 (Zeng et al., 2014). Here, NFKB1 is linked to melanoma.