To understand the molecular mechanisms by which miR‐193a mediates the functions of NEAT1 in CRC cells, miR‐193a inhibitor and sh‐NEAT1 were co‐transfected into LOVO and HCT116 cell lines, indicating that miR‐193a inhibitor promoted cell proliferation and invasive potential, but knockdown of NEAT1 counteracted these tumour promoting effects induced by miR‐193a inhibitor (Figure 5A,B). The gene discussed is NEAT1; the disease is colorectal carcinoma.