PDLIM7 and nasopharyngeal carcinoma: Latent membrane protein 1 (LMP1) of Epstein-Barr virus (EBV) inactivates LKB1/AMPK, whereas AMPK activation by AICAR abrogated LMP1-mediated proliferation and transformation of nasopharyngeal epithelial cells, suggesting therapeutic potential for EBV-associated nasopharyngeal carcinoma [42].