Second, in dystonia musculorum in mice (that corresponds to the hereditary sensory autonomic neuropathy type VI [HSAN-VI] in humans [38]), mutations in the dystonin (DST) gene disrupt the organization of the Golgi apparatus, stability of the microtubules, and transport of secretory molecules through the cell [39, 40]. The gene discussed is DST; the disease is hereditary sensory and autonomic neuropathy type 6.