EGCG selectively inhibits the expression of vascular endothelial growth factor VEGF-C (VEGFC) and the tyrosine kinase receptor, VEGFR2, by inhibition of the VEGFC/VEGFR2 signaling pathways, which inhibits endometriosis-associated angiogenesis of endothelial cells [72]. This evidence concerns the gene KDR and endometriosis.