Based on this, we decided to examine whether the response to the Th17 subpopulation induced by capsular GalXM-stimulated dendritic cells is related to protection in the murine model during experimental cryptococcosis, despite Zelante and colleagues98 having demonstrated that in fungal infections with C. albicans and Aspergillus fumigatus, an IL-23- and IL-17-mediated response subverted the inflammatory neutrophil program, resulting in severe inflammatory tissue pathology associated with the infections. Here, IL17A is linked to fungal infectious disease.