Previous work with C. elegans has identified defects in calcium signaling and acetylcholine sensitivity as pathophysiologies associated with dystrophin deficiency, so it is possible that these defects are more severe in dys-1(eg33) than in dys-1(cx18) (Mariol and Ségalat, 2001; Zhan et al., 2014; Bessou et al., 1998; Giugia et al., 1999). Here, DMD is linked to hyperinsulinemic hypoglycemia, familial, 4.