GCTB is characterized by a high recurrence rate, and studies have shown that aberrantly highly expressed β-catenin and IL-17A are closely associated with GCTB progression, including tumor growth and recurrence.3, 4 Thus, understanding the biological basis for the deregulation of β-catenin and IL-17A is of great value for the future development of novel therapeutic strategies. Here, IL17A is linked to neoplasm.