We report somatic, heterozygous mutations in 72% of GCLJ in three genetic drivers: TRPV4, KRAS, and FGFR1. It is possible that our data underestimates the proportion of samples carrying a mutation in any of these 3 genes, as 11 samples were solely analyzed using standard sequencing, which may miss cases with lower mutational reads based on regional contamination with tumor microenvironment. This evidence concerns the gene FGFR1 and neoplasm.