EGFR and gastritis: Five putative targets of ZJW (interleukin-6[IL-6], IL-1β, tumor necrosis factor [TNF]-α, monocyte chemotactic protein [MCP]-1 and EGFR), and two known therapeutic targets of gastritis (cholecystokinin-B receptors[CCKBR] and IL-12β) and a link target between ZJW and disease NF-κB are shown to be the most active factors that participate in these key pathways, which are implied an important role in the occurrence and promotion of gastritis.