However, Kumei et al., using a diet‐induced nonalcoholic steatohepatitis model, found that deficiency in IP receptors accelerates disease progression by augmenting histological derangements including cell infiltration accompanied by increased expression of CCL‐2 (also called MCP‐1) and TNF‐α. Here, CCL2 is linked to metabolic dysfunction-associated steatohepatitis.