Similarly, the evolving ovarian cancer metastatic tumors are metabolically reprogrammed by CAFs through the secretion of C-C Motif Chemokine Ligand 5 (CCL5), C-X-C motif chemokine ligand 10 (CXCL10), and IL-6 to utilize glycogen [38]. This evidence concerns the gene CCL5 and metastatic neoplasm.