Interestingly, markers within KIAA0319 and FOXP2 were significantly related to dyslexia when comorbid samples were included (Dys+Com-vs-Ctr_Dys), as well as when comorbids were analyzed as an independent group (Com-vs-Ctr_Dys/Com-vs-Ctr_ADHD/Com-vs-Ctr_Com). This evidence concerns the gene KIAA0319 and attention deficit-hyperactivity disorder.