This is consistent with results from Terra et al., who reported a lack of correlation between hepatic expression of RBP4 and systemic levels in controls and in morbidly obese women with and without NAFLD.[26] In summary, these findings suggest that other causes may disrupt the relation between retinol and RBP4, or, alternatively, hepatic RBP4 expression does not translate into serum RBP4 levels. Here, RBP4 is linked to metabolic dysfunction-associated steatotic liver disease.