GAP43 and myocardial infarction: In addition, in a canine model of MI, increased nerve activity associated with increased neuronal size and synaptic density in bilateral SGs was reported.38 A similar change was observed in a porcine model of MI.39 These results provided a possible mechanistic link between neural remodeling and VA.40 In our study, we assessed sympathetic neural remodeling by TH and GAP‐43 immunoreactivity and concluded that RDN reversed sympathetic neural remodeling in the IBZ and bilateral SGs.