Administration of the site-1/2b anti-IL-7Rα mAbs dramatically prevented memory T cell-mediated skin inflammation after antigen rechallenge as displayed by a marked reduced erythema response (Fig. 2a–c) and immune cell skin infiltration (Fig. 2b) in all baboons (n = 3 with IgG1 mAb and n = 3 with IgG4 mAb). This evidence concerns the gene IL7R and Erythema.