Many other enzymes directly implicated in sulfur metabolism (APS, SIR, MetS, and OAS) or related to it (γECS and several isoforms of Gpx, Grx, and GST) were found also to be nitrosylated in mutant plants (gsnor1-3) defective in GSNOR (Hu et al., 2015), clearly evidencing a central role of GSNOR in nitrosothiol homeostasis. Here, ADH5 is linked to metabolic syndrome.