Additional gain‐ and loss‐of‐function analyses indicated that NEAT1 modulated CCI‐induced neuropathic pain by regulating the miR‐381/HMGB1 axis.51 These results suggested that NEAT1 and HMGB1 are potential therapeutic targets in neural compression‐induced neuropathic pain (Table 2). The gene discussed is HMGB1; the disease is neuropathic pain.