SLC10A1 and infection: We infected differentiated spheres at days 0 and 10 with HBV-containing sera at a multiplicity of infection (MOI) of 300 in the absence or presence of entecavir (ETV), a reverse-transcription inhibitor or tauroursodeoxycholic acid (TUDC),14 a substrate of NTCP that inhibits HBV pre-S1 lipopeptide binding.