RMDN3 and dysplastic nevus: These findings are corroborated by the interaction profile of PTPIP51 and PTP1B, the phosphatase which is responsible for dephosphorylation of tyrosine 176, with the highest number of interactions in normal melanocytes and lowest number of interactions in melanocytes from dysplastic nevi and an interaction profile laying in between the aforementioned for malignant melanocytes.